Pro-inflammatory CX3CR1+ CD59+ TL1A+ IL-23+ monocytes are expanded in patients with Ankylosing Spondylitis and modulate ILC3 immune functions

Francesco Dieli; Diana Di Liberto; Laura Saieva; Francesco Ciccia; Riccardo Alessandro; Giuliana Guggino; Laura Saieva; Nigil Haroon; Ranjeny Thomas; Vidya Ranganathan; Arifur Rahman; Francesco Dieli; Michael Zeng; Francesco Dieli; Arifur Rahman; Diana Di Liberto; Riccardo Alessandro; Dominique Baeten; Roberto Giacomelli; Paola Cipriani

Pro-inflammatory CX3CR1+ CD59+ TL1A+ IL-23+ monocytes are expanded in patients with Ankylosing Spondylitis and modulate ILC3 immune functions

Francesco Dieli, Diana Di Liberto, Laura Saieva, Francesco Ciccia, Riccardo Alessandro, Giuliana Guggino, Laura Saieva, Nigil Haroon, Ranjeny Thomas, Vidya Ranganathan, Arifur Rahman, Francesco Dieli, Michael Zeng, Francesco Dieli, Arifur Rahman, Diana Di Liberto, Riccardo Alessandro, Dominique Baeten, Roberto Giacomelli, Paola Cipriani

Risultato della ricerca: Article › peer review

26 Citazioni (Scopus)

Abstract

Gut derived ILC3 have been demonstrated to participate in AS pathogenesis. CX3CR1+ mononuclear phagocytes (MNP) have been demonstrated to modulate ILC3 function in the gut. The aim of this study was to study the role of pro-inflammatory CX3CR1+ CD59+ MNP in modulating ILC3 function in AS patients.

Lingua originale	English
Numero di pagine	15
Rivista	ARTHRITIS & RHEUMATOLOGY
Stato di pubblicazione	Published - 2018

All Science Journal Classification (ASJC) codes

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Dieli, F., Di Liberto, D., Saieva, L., Ciccia, F., Alessandro, R., Guggino, G., Saieva, L., Haroon, N., Thomas, R., Ranganathan, V., Rahman, A., Dieli, F., Zeng, M., Dieli, F., Rahman, A., Di Liberto, D., Alessandro, R., Baeten, D., Giacomelli, R., & Cipriani, P. (2018). Pro-inflammatory CX3CR1+ CD59+ TL1A+ IL-23+ monocytes are expanded in patients with Ankylosing Spondylitis and modulate ILC3 immune functions. ARTHRITIS & RHEUMATOLOGY.

Dieli, F , Di Liberto, D , Saieva, L , Ciccia, F , Alessandro, R , Guggino, G, Saieva, L, Haroon, N, Thomas, R, Ranganathan, V, Rahman, A, Dieli, F, Zeng, M, Dieli, F, Rahman, A, Di Liberto, D, Alessandro, R, Baeten, D, Giacomelli, R & Cipriani, P 2018, 'Pro-inflammatory CX3CR1+ CD59+ TL1A+ IL-23+ monocytes are expanded in patients with Ankylosing Spondylitis and modulate ILC3 immune functions', ARTHRITIS & RHEUMATOLOGY.

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title = "Pro-inflammatory CX3CR1+ CD59+ TL1A+ IL-23+ monocytes are expanded in patients with Ankylosing Spondylitis and modulate ILC3 immune functions",

abstract = "Gut derived ILC3 have been demonstrated to participate in AS pathogenesis. CX3CR1+ mononuclear phagocytes (MNP) have been demonstrated to modulate ILC3 function in the gut. The aim of this study was to study the role of pro-inflammatory CX3CR1+ CD59+ MNP in modulating ILC3 function in AS patients.",

keywords = "CX3CR1+ monocytes, IL-23, ILC3, TL1A, gut inflammation, CX3CR1+ monocytes, IL-23, ILC3, TL1A, gut inflammation",

author = "Francesco Dieli and {Di Liberto}, Diana and Laura Saieva and Francesco Ciccia and Riccardo Alessandro and Giuliana Guggino and Laura Saieva and Nigil Haroon and Ranjeny Thomas and Vidya Ranganathan and Arifur Rahman and Francesco Dieli and Michael Zeng and Francesco Dieli and Arifur Rahman and {Di Liberto}, Diana and Riccardo Alessandro and Dominique Baeten and Roberto Giacomelli and Paola Cipriani",

year = "2018",

language = "English",

journal = "ARTHRITIS & RHEUMATOLOGY",

issn = "2326-5191",

}

TY - JOUR

T1 - Pro-inflammatory CX3CR1+ CD59+ TL1A+ IL-23+ monocytes are expanded in patients with Ankylosing Spondylitis and modulate ILC3 immune functions

AU - Dieli, Francesco

AU - Di Liberto, Diana

AU - Saieva, Laura

AU - Ciccia, Francesco

AU - Alessandro, Riccardo

AU - Guggino, Giuliana

AU - Saieva, Laura

AU - Haroon, Nigil

AU - Thomas, Ranjeny

AU - Ranganathan, Vidya

AU - Rahman, Arifur

AU - Dieli, Francesco

AU - Zeng, Michael

AU - Dieli, Francesco

AU - Rahman, Arifur

AU - Di Liberto, Diana

AU - Alessandro, Riccardo

AU - Baeten, Dominique

AU - Giacomelli, Roberto

AU - Cipriani, Paola

PY - 2018

Y1 - 2018

N2 - Gut derived ILC3 have been demonstrated to participate in AS pathogenesis. CX3CR1+ mononuclear phagocytes (MNP) have been demonstrated to modulate ILC3 function in the gut. The aim of this study was to study the role of pro-inflammatory CX3CR1+ CD59+ MNP in modulating ILC3 function in AS patients.

AB - Gut derived ILC3 have been demonstrated to participate in AS pathogenesis. CX3CR1+ mononuclear phagocytes (MNP) have been demonstrated to modulate ILC3 function in the gut. The aim of this study was to study the role of pro-inflammatory CX3CR1+ CD59+ MNP in modulating ILC3 function in AS patients.

KW - CX3CR1+ monocytes

KW - IL-23

KW - ILC3

KW - TL1A

KW - gut inflammation

KW - CX3CR1+ monocytes

KW - IL-23

KW - ILC3

KW - TL1A

KW - gut inflammation

UR - http://hdl.handle.net/10447/295625

M3 - Article

SN - 2326-5191

JO - ARTHRITIS & RHEUMATOLOGY

JF - ARTHRITIS & RHEUMATOLOGY

ER -

Pro-inflammatory CX3CR1+ CD59+ TL1A+ IL-23+ monocytes are expanded in patients with Ankylosing Spondylitis and modulate ILC3 immune functions

Abstract

All Science Journal Classification (ASJC) codes

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